Present concept of central serous chorioretinopathy classification does not reflect the true picture of the pathological process. The purpose of the study was the development of diagnostic criteria of central serous chorioretinopathy clinical forms and basing on that extend and optimize its classification with assessment of risk factors and prognosis of disease. Full ophthalmologic and somatic examination of 56men with central serous chorioretinopathy was made. Focal laser coagulation was performed in patients with fluorescein leakage and local injury of pigment epithelium by angiographic data (22patients). In the case of extended damage of pigment epithelium and diffuse leakage of fluorescein (23people) transpupillary thermotherapy of optical disk was conducted. Detailed expert evaluation of retinal changes by fluorescein angiographic data allowed dividing the patients into 3groups. The main criterion for the division was the damaged area of the pigment epithelium: group 1 – central serous chorioretinopathy with local injury of pigment epithelium (with leakage point); group 2 –central serous chorioretinopathy with extensive damage of pigment epithelium with diffuse leakage; group 3 –central serous chorioretinopathy with extensive damage of pigment epithelium combined with leakage point (recurrence of leakage).The results of our study brought us to the following conclusion. Development of new classification criteria of the disease allows to construct a more specific algorithm of therapeutic measures and to determine prognosis of central serous chorioretinopathy already at the stage of diagnostics.
central serous chorioretinopathy, retinal detachment of the neuroepithelium, detachment of retinal pigment epithelium, classification
1. VolkovaNV, ShchukoAG, MalyshevVV (2010). Retrospective analysis of risk factors for the develop-ment of scar changes the intraocular fluid outflow pathways after fistulizing antiglaucomatous surgery. Report 1 [Retrospektivnyy analiz faktorov riska razvitiya rubtsovykh izmeneniy putey ottoka vnutriglaznoy zhidkosti posle fistuliziruyushchikh antiglaukomatoznykh operatsiy. Soobshchenie 1]. Natsional’nyy zhurnal Glaukoma, (3), 35-40.
2. VolkovaNV, ShchukoAG, NoskovaLK, MalyshevVV(2011). Role of hyperlipoperoxidation and initial hormonal regulation in the mechanisms of scarring changes of intraocular fluid outflow pathways after fistulizing antiglaucomatous surgery. Report 2 [Rol’ giperlipoperoksidatsii i iskhodnoy gormonal’noy regulyatsii v mekhanizmakh rubtsovykh izmeneniy putey ottoka vnutriglaznoy zhidkosti posle fistuliziruyushchikh antiglaukomatoznykh operatsiy. Soobshchenie 2]. Natsional’nyy zhurnal Glaukoma, 10(2). 3-7.
3. KatznelsonLA, ForofonovaTI, BuninAY (1990). Vascular diseases of the eyes [Sosudistye zabolevaniya glaz], 193
4. GassJD (1991). Central serous chorioretinopathy and white subretinal exudation during pregnancy. Arch. Ophthalmol., (109), 677-681.
5. GuyerDR, YannuzziLA, SlakterJS, SorensonJA,HoA, OrlockD (1994). Digital indocyanine green videoangiography of central serous chorioretinopathy. Arch. Ophthalmol., (112), 1057-1062.
6. IidaT, KishiS, HagimuraN, ShimizuK (1999). Persistent and bilateral choroidal vascular abnormalities in central serous chorioretinopathy. Retina, (19), 508-512.
7. ImamuraY, FujiwaraT, MargolisR, SpaideRF (2009).Enhanced depth imaging optical coherence tomography of the choroid in central serous chorioretinopathy. Retina, (29), 1469-1473.
8. MarmorMF (1988). New hypothesis on the pathogenesis and treatment of serous retinal detachment. Graefes Arch. Clin. Exp. Ophthalmol., (226), 548-552.
9. MoschosM, BrouzasD, KoutsandreaC, StefanosB, LoukianouH, PapantonisF, MoschosM (2007). Assess-ment of central serous chorioretinopathy by optical co-herence tomography and multifocal electroretinography. Ophthalmologica, 221(5), 292-298.
10. SekiryuT, IidaT, MarukoI, SaitoK, KondoT (2010). Infrared fundus autofluorescence and central serous chorioretinopathy. Investigative Ophthalmology & Visual Science, 51(10), 4956-4962.
11. YannuzziLA (1986). Type A behavior and central serous chorioretinopathy. Trans. Am. Ophthalmol. Soc., (84) 799-845
